Aryl hydrocarbon receptor (AhR)-mediated interactions between human follicular thyroid cells and monocytes.
Inflammation associated with microbial pathogens is thought to play a key role in the pathogenesis of thyroid nodules. The contribution of bacterial antigens, Toll-like receptor 4 (TLR4), and its activated receptor, myeloid differentiation protein-2, have been linked to thyroid pathogenesis. Because prior investigations have focused on the effects of TLR4 on thyroid 2336c5e09f
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